Now this is unconfirmed, but it sounds a lot like the story of the workers who got sick and died in a day of acute lukemia for no apparent reason since there wasn't enough evidence to prove radiation had anything to do with it. There is a clinical condition called radiation nephropathy which is kidney failure due to too much ionizing radiation.
Here's what enenews reported:
Here's what enenews reported:
Published: November 13th, 2011 at 10:55 AM EDT By ENENEWS Staff
Report: Fireman dies after working in Fukushima after quake — “Vomited blood frequently and finally died of renal failure”
Uploaded by: tokyobrowntabby
Upload Date: Nov 10, 2011
This video clip is from the Q&A session after the lecture by an actor-and-anti-nuke-activist Taro Yamada (man on the right), at the “National Forum of School Lunch” held in Sapporo City on November 6、2011.Transcript Excerpts
The death of the member of the special rescue unit of the Fire Department, whom the questioner in the video is talking about, hasn’t been confirmed officially nor reported in MSM.
The person called “Dr. Sakiyama” in the video is Dr. Hisako Sakiyama, a specialist in radiation exposure at Takagi School and ex-chief researcher at National Institute of Radiological Sciences. She was a researcher at MIT before working for the Institute.
Translation and captioning by tokyobrowntabby.
On October 26, a friend of mine in Osaka passed away.About renal failure after a nuclear catastrophe (via study):
He was a rescue squad member and had been sent to work in disaster-affected areas for a long time, such as Iwate or Fukushima.
In July, he was found to have been internally exposed to radiation.
All his team members had been, too.
But their mission didn’t end. [...]
Eventually they got sick and realized they couldn’t continue their duties any more.
All the team members including him quit the rescue squad. Before they quit, they had been berated by their supervisors as unpatriotic.
In a little more than 3 months since his internal exposure was found in July, my friend vomited blood frequently and finally died of renal failure.
“The rate of extragenital pathology (problems other than reproductive system?) during pregnancy increased in the years after the accident from 23.1% in 1982 to 33.9% in 1990 in Chechersky and from 7.1% to 51.2% in Polessky over the same period (Fig. 2). The main pathologies in both districts were anemia of pregnancy, renal disorders, transient hypertension, and abnormalities of fat metabolism.
What is renal failure:
You +1'd this publicly. UndoRenal failure or kidney failure (formerly called renal insufficiency) describes a medical condition in which the kidneys fail to adequately filter toxins and waste ...
Renal Failure and radiation:
Radiation nephropathy is kidney injury and impairment of function caused by ionizing radiation. It may occur after irradiation of one or both kidneys, and it may result in kidney failure.
Classic radiation nephropathy occurs after bilateral, local kidney irradiation. It is a syndrome of chronic renal failure, occurring months or years after renal irradiation. Acute radiation nephropathy develops 6-12 months after irradiation, whereas chronic radiation nephropathy develops years later. Radiation nephropathy has also been discovered to cause chronic renal failure after bone marrow transplantation (BMT). In addition, the use of yttrium–90–tagged (90 Y-tagged) somatostatin and other radionuclides for radionuclide therapy cause radiation nephropathy when they are filtered by the kidneys and reabsorbed by the renal tubule epithelium or when blood-borne exposure to the kidney cells occurs. (See Etiology,
British Journal of Radiology (2005) Supplement_27, 82-88
© 2005 British Institute of Radiology
Radiation-induced multi-organ involvement and failure: the contribution of radiation effects on the renal system
Clinical and experimental studies suggest that acute or chronic renal injury could occur after certain types of radiation accidents. Such renal injury could be life-threatening in its own right, could exacerbate other radiation injuries and could complicate the treatment of non-radiation injuries. The clinical experience with therapeutic bone marrow transplantation (BMT) indicates that acute renal failure is conceivable in radiation accident victims who receive BMT, and there are experimental data that suggest that radiation-induced bone marrow aplasia could exacerbate acute renal failure caused by other agents. Both the experimental data and the clinical experience with therapeutic BMT also suggest that chronic renal failure could occur after radiation accidents if the bilateral renal dose exceeded 4–5 Gy and if bone marrow toxicity was avoided by partial body shielding or therapeutic interventions. In addition, clinical experience using radiolabelled biologicals in cancer therapy shows that internal deposition of certain types of radioactive material can cause chronic renal failure. There is clinical evidence that the progression of established chronic radiation-induced renal injury can be attenuated by treatment with angiotensin converting enzyme (ACE) inhibitors or angiotensin II (AII) receptor antagonists. There is also pre-clinical evidence that the risk of radiation-induced chronic renal failure can be reduced through prophylactic use of ACE inhibitors or AII receptor antagonists, and a randomised clinical trial of this approach is in progress.